ScienceDaily (Apr. 27, 2009) — Some DNA repair enzymes can become double-edged swords – If they work too slowly, they can block necessary cell maintenance and contribute to cell death. This could explain the somewhat mysterious success of the widely used cancer drug 5-Fluorouracil (5FU) and help clinicians to predict patient’s response to chemotherapy, according to new findings from the University of Basel, Switzerland.
