The immune system’s response against amyloid-beta, the protein that forms plaques in the brains of patients with Alzheimer’s disease, appears to protect the brain from damage in early stages of the devastating neurological disorder. A report from Massachusetts General Hospital (MGH) researchers finds that lack of a protein required for recruitment of the brain’s primary immune cell led to increased amyloid-beta deposits and earlier death in a mouse model of Alzheimer’s disease. The paper will appear in the journal Nature Medicine and has received early online release. >>> Discuss This Story
