Neuron Loss in Schizophrenia and Depression Could be Prevented With an Antioxidant, Biological Psychiatry Reveals

Gamma-aminobutyric acid (GABA) deficits have been implicated in schizophrenia and depression. In schizophrenia, deficits have been particularly well-described for a subtype of GABA neuron, the parvalbumin fast-spiking interneurons. The activity of these neurons is critical for proper cognitive and emotional functioning.

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